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苯芴酮號：975-17-7   
英文名稱：Phenylfluorone；2,6,7-Trihydroxy-9-phenylisoxanthen-3-one；Florone black；9-Phenyl-2,6,7-trihydroxy-3

-fluorone；2,6,7-Trihydroxy-9-phenyl-3H-xanthen-3-one；9-Phenyl-2,3,7-trihydroxy-6-fluorone 2,3,7-Trihydroxy-9-

phenylxanthen-6-one；2,6,7-Trihydroxy-9-phenylfluor   
其他名稱：鍺試劑；苯基芴酮；苯基熒光酮；2,6,7-三羥基-9-苯基熒光酮；2,6,7-三羥基-9-苯基-3H-呫噸-3-酮；9-苯基-2,3,7-三羥

基-6-氧蒽酮；2,6,7-三羥基-9-苯基-6-熒光酮；9-苯基-2,3,7-三羥基-6-熒光酮   
號：975-17-7   
C19H12O5=320.30   
級別：AR   
乙醇溶解試驗：合格   
對鍺靈敏度試驗：合格   
灼燒殘渣(以硫酸鹽計)：≤0.1%   
性狀(以下信息僅供參考)：橙紅色結晶性粉末。易溶于堿，溶于乙醇鹽酸和乙醇硫酸的混合液，微溶于乙醇，不溶于水、苯和方。熔

點>300℃。Z大吸收波長 552nm。   
用途：本品僅供科研，不得用于其它用途。(以下用途僅供參考)光度測定鍺、鉬、鈮、銻、錫和鉭等。   
保存：RT，避光

客戶根據苯芴酮號：975-17-7性質、化學式、分子式、結構式、比重、密度、號、沸點、熔點、水溶性、MSDS、用途、作用、規格包裝、性狀、注意事項、英文名、別稱、純度、級別等情況，本產品化學性質穩定，運輸條件不苛刻，一般儲存在陰涼，干燥，通風良好的地方，遠離不相容的物質。保持容器密閉。
試劑品牌：TCI、sigma、Alfa、Avocado、Aldrich、ACROS、Fluka、ICN（MP）等
包裝：1kg、100g、10g、250g、25g、500g、50g、5g等包裝
級別：GR級別、AR級別、CP級別、L.P.級別等
公司提供的苯芴酮號：975-17-7*，貨源充足。嚴格的生產質量控制體系，包括：優級純，分析純，化學純，試劑級，基準試劑，實驗純，教學試劑，高純試劑，色譜純，光譜純，電子純。各種包裝規格，并可提供包裝定制，咨詢訂購。

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【服務宗旨】
竭誠提供優質產品，售后服務客戶滿意度100％
交叉反應  Human, Mouse, Rat, Chicken, Dog, Pig, Cow
產品類型  一抗  磷酸化抗體  
研究領域  腫瘤 免疫學 信號轉導 轉錄調節因子 激酶和磷酸酶 
蛋白分子量  predicted molecular weight: 105kDa
性    狀  Lyophilized or Liquid
免 疫 原  KLH conjugated Synthesised phosphopeptide derived from human NF KappaB p105 around the phosphorylation site of Thr931
亞    型  IgG
純化方法  affinity purified by Protein A
儲 存 液  0.01M PBS, pH 7.4 with 10 mg/ml BSA and 0.1% Sodium azide
產品應用   WB=1:100-500  ELISA=1:500-1000  IP=1:20-100  IHC-P=1:100-500  IHC-F=1:100-500  IF=1:100-500
（石蠟切片需做抗原修復）
 not yet tested in other applications.
 optimal dilutions/concentrations should be determined by the end user. 
保存條件  Store at -20 °C for one year. Avoid repeated freeze/thaw cycles. The lyophilized antibody is stable at room temperature for at least one month and for greater than a year when kept at -20°C. When reconstituted in sterile pH 7.4 0.01M PBS or diluent of antibody the antibody is stable for at least two weeks at 2-4 °C.
Important Note  This product as supplied is intended for research use only, not for use in human, therapeutic or diagnostic applications.
產品介紹 This gene encodes a 105 kD protein which can undergo cotranslational processing by the 26S proteasome to produce a 50 kD protein. The 105 kD protein is a Rel protein-specific transcription inhibitor 苯芴酮號：975-17-7and the 50 kD protein is a DNA binding subunit of the NF-kappa-B (NFKB) protein complex. NFKB is a transcription regulator that is activated by various intra- and extra-cellular stimuli such as cytokines, oxidant-free radicals, ultraviolet irradiation, and bacterial or viral products. Activated NFKB translocates into the nucleus and stimulates the expression of genes involved in a wide variety of biological functions. Inappropriate activation of NFKB has been associated with a number of inflammatory diseases while persistent inhibition of NFKB leads to inappropriate immune cell development or delayed cell growth. SUBUNIT: Active NF-kappa-B is a heterodimer of an about 50 kDa DNA-binding subunit and the weak DNA-binding subunit p65. Two heterodimers might form a labile tetramer. Also interacts with MAP3K8. NF-kappa-B p50 subunit interacts with NCOA3 coactivator, which may coactivate NF-kappa-B dependent expression via its histone acetyltransferase activity. Interacts with DSIPI; this interaction prevents nuclear translocation and DNA-binding. Interacts with SPAG9 and UNC5CL.
Function : NF-kappa-B is a pleiotropic transcription factor present in almost all cell types and is the endpoint of a series of signal transduction events that are initiated by a vast array of stimuli related to many biological processes such as inflammation, immunity, differentiation, cell growth, tumorigenesis and apoptosis. NF-kappa-B is a homo- or heterodimeric complex formed by the Rel-like domain-containing proteins RELA/p65, RELB, NFKB1/p105, NFKB1/p50, REL and NFKB2/p52 and the heterodimeric p65-p50 complex appears to be most abundant one. The dimers bind at kappa-B sites in the DNA of their target genes and the individual dimers have distinct preferences for different kappa-B sites that they can bind with distinguishable affinity and specificity. Different dimer combinations act as transcriptional activators or repressors, respectively. NF-kappa-B is controlled by various mechanisms of post-translational modification and subcellular compartmentalization as well as by interactions with other cofactors or corepressors. NF-kappa-B complexes are held in the cytoplasm in an inactive state complexed with members of the NF-kappa-B inhibitor (I-kappa-B) family. In a conventional activation pathway, I-kappa-B is phosphorylated by I-kappa-B kinases (IKKs) in response to different activators, subsequently degraded thus liberating the active NF-kappa-B complex which translocates to the nucleus. NF-kappa-B heterodimeric p65-p50 and RelB-p50 complexes are transcriptional activators. The NF-kappa-B p50-p50 homodimer is a transcriptional repressor, but can act as a transcriptional activator when associated with BCL3. NFKB1 appears